- **Self-sampling of bodily fluids**: Participants discussed which non-blood fluids can be extracted with needles/syringes at home; lymphatic fluid, saliva, semen and sweat were suggested, and the term “nonblood specimens” from phlebotomy guidelines was noted as a starting point for best-practice information.

- **Androgen receptor gene as cognitive modifier**: A paper proposing that polymorphisms in the androgen-receptor gene act as a “major modifier” of neuronal transmission speed and intelligence was shared (https://www.sciencedirect.com/science/article/pii/S030698770600658X).

- **Ecological drivers of primate intelligence**: A study arguing that degree of home-range overlap among primates is a key ecological pressure driving the evolution of intelligence was circulated (https://onlinelibrary.wiley.com/doi/full/10.1002/ajp.22357).

- **Brain size vs. neuron count trade-off**: Research showing that in mice larger brains do not contain more neurons because increased cell number is offset by reduced average cell size was highlighted (https://www.frontiersin.org/articles/10.3389/fnana.2015.00064/full).

- **Dopamine D1 receptor dynamics and general cognitive ability (GCA)**: Multiple papers were posted demonstrating that (a) D1 receptor density in the medial prefrontal cortex rises within 60 min of high working-memory demand, (b) innate GCA in mice correlates with D1 receptor turnover rate, and (c) DRD1 mRNA levels predict cognitive performance (https://www.nature.com/articles/s41598-018-22668-0, https://www.nature.com/articles/nn1846).

- **“Flow proneness” linked to D2 receptor availability**: A human imaging study finding that individual differences in the propensity to experience “flow” correlate with dopamine D2-receptor availability in the dorsal striatum—and that the DRD2 C957T polymorphism is a genetic marker for this trait—was added to the wiki (https://www.sciencedirect.com/science/article/pii/S1053811912010762).

- **Skepticism about dopamine-centric explanations**: Channel members expressed caution toward over-attributing cognitive or behavioral phenomena to simple “dopamine (or serotonin) imbalance” narratives, noting widespread hype around the dopamine/serotonin axis.